Rheumatoid A chronic autoimmune systemic illness of an inflammatory origin, arthritis (RA) affects synovial joints as well as other parts of the body [1]. According to earlier research [2], RA affects 39.19 percent of the global population.
In addition to its joint manifestations, RA may have extraarticular manifestations in any organ system, and these manifestations may occur prior to the development of arthritis itself. Entrapment neuropathy, a complication of proliferative synovitis, is the most prevalent neurological symptom of rheumatoid arthritis (3).
Neurological issues affect around a third of individuals throughout their lifetimes
3.
Compressive neuropathy, distal sensory neuropathy, and mixed sensorimotor neuropathy are the most prevalent types of peripheral nerve involvement in rheumatoid arthritis (4).
Neuropathy in rheumatoid arthritis may be caused by vasculitis with typical axonal loss and accumulation of immune complexes and the fixation of complement5. The walls of the arteries show fibrinoid type arteritis and immunological globins as symptoms (6).
Individuals with rheumatoid arthritis may confuse neurological symptoms with those of joint illness, making it difficult to detect the existence of peripheral neuropathy in these patients. When a patient has a serious joint condition, it is difficult to evaluate the neurological system (5). Thus, our present research is aimed at assessing the incidence and pattern of neuropathy in rheumatoid arthritis and correlating it with clinical parameters and other extraarticular involvement (2).
To evaluate neuropathic pain in individuals with rheumatoid arthritis (RA) who are experiencing neuropathic symptoms. 2. Objective.
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